Histophilus somni [recurso electrónico] : Biology, Molecular Basis of Pathogenesis, and Host Immunity / edited by Thomas J. Inzana.
Tipo de material: TextoSeries Current Topics in Microbiology and Immunology ; 396Editor: Cham : Springer International Publishing : Imprint: Springer, 2016Descripción: VII, 160 p. online resourceTipo de contenido: text Tipo de medio: computer Tipo de portador: online resourceISBN: 9783319295565Tema(s): Medicine | Medical microbiology | Veterinary medicine | Bacteriology | Biomedicine | Medical Microbiology | Veterinary Medicine | BacteriologyFormatos físicos adicionales: Printed edition:: Sin títuloClasificación CDD: 616.9041 Clasificación LoC:QR46Recursos en línea: Libro electrónicoTipo de ítem | Biblioteca actual | Colección | Signatura | Copia número | Estado | Fecha de vencimiento | Código de barras |
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Libro Electrónico | Biblioteca Electrónica | Colección de Libros Electrónicos | 1 | No para préstamo |
Taxonomy of Histophilus somni -- Histophilosis as a Natural Disease -- Histophilus somni Genomics and Genetics -- Interactions on Histophilus somni with Host Cells -- Histophilus somni Surface Proteins -- Host Immune Response to Histophilus somni -- The Many Facets of Lipooligosaccharide as a Virulence Factor of Histophilus somni -- Exopolysaccharide Production and Biofilm Formation by Histophilus somni.
This volume reviews the current understanding of the taxonomy, disease syndromes, genetics, biology, and pathogenic factors of Histophilus somni, as well as the host immune response to this pathogen. H. somni is one of the most important bacterial pathogens in cattle and other ruminants, and its virulence factors are highly conserved with Haemophilus influenzae and other members of the Pasteurellaceae. H. somni has been recognized as a major cause of thrombotic meningoencephalitis, respiratory disease syndromes, myocarditis, reproductive disease syndromes, polyarthritis, mastitis, ocular disease, and septicemia. The only known habitats of H. somni are the mucosal surfaces of ruminants, making this bacterium an opportunistic pathogen. Although it is capable of causing inflammation at systemic sites and is toxic to epithelial and phagocytic cells, the bacterium?s wide array of virulence factors act primarily as a defense against, or to escape recognition from, host innate and adaptive immunity.